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. 2023 Jan 2;42:1. doi: 10.1186/s13046-022-02574-0

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Correlation between ENO2 expression level and HNSCC development. a Analysis of ENO2 expression in normal and tumor tissues using TCGA cohort with 33 cancer types. b The expression levels of ENO2 in head and neck normal and tumor tissues using GEO dataset. c Kaplan-Meier survival analysis of the correlation between the expression levels of ENO2 and survival in HNSCC patients. d Chi-square test analysis of the correlation between the expression levels of ENO2 and clinical stage of HNSCC patients. e, f ENO2 expression levels in human primary HNSCC samples (n=52) and normal tissues (n=19) determined by IHC. Representative IHC result and quantitative data are shown in e and f, respectively. g, h Transcriptional (g) and protein levels (h) of ENO2 in different HNSCC cell lines determined by qRT-PCR and western blotting, respectively. **p<0.01