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. 2023 Jan 4;20:4. doi: 10.1186/s12974-022-02674-w

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — tMCAO leads to higher NLRP3 levels 24 h after stroke induction compared to sham mice. NLRP3 protein expression in cortex and basal ganglia of vehicle- and sham treated mice on day 1 after tMCAO. For densitometric quantification actin was used as a loading control (n ≥ 7). Data was analyzed by unpaired t test. *p < 0.05