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. 2022 Oct 31;17(1):150–172. doi: 10.1002/1878-0261.13324

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© 2022 The Authors. Molecular Oncology published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 5 — Genomic alterations in 10 telomerase components across cancer types. (A, B) Amplifications and homozygous deletions of 10 telomerase components across cancer types, respectively. The analyzed patient number was 9910. (C) The landscape of overall genomic alterations in 10 telomerase components across 33 cancer types. The analyzed patient number was 10 245. (D–F) Differential genomic alterations of telomerase components in three cancer types. The analyzed patient numbers were 533, 486, and 493 for uterine corpus endometrial carcinoma (UCEC), lung squamous cell carcinoma (LUSC), and prostate adenocarcinoma (PRAD), respectively.