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. 2022 Nov 22;36(1):209–222. doi: 10.1007/s13577-022-00820-3

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Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — TRIM59 overexpression exacerbates PA-induced steatosis and inflammation in L02 cells. A, B The expression of TRIM59 in L02 cells transduced with overexpressed TRIM59 lentivirus (oeTRIM59) with the presence of palmitic acid (PA) or not. C Oil red O staining detected the level of steatosis in L02 cells treated with oeTRIM59and 0.4 mM PA (magnification: 200×). E The level of TNF-α, IL-6, and IL-8 in L02 cells treated with oeTRIM59 and 0.4 mM PA. *P < 0.05, **P < 0.01, ***P < 0.001, n = 3