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. 2022 Dec 15;34(1):ar7. doi: 10.1091/mbc.E22-05-0176

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© 2023 Perez-Vale, Yow, et al. “ASCB®,” “The American Society for Cell Biology®,” and “Molecular Biology of the Cell®” are registered trademarks of The American Society for Cell Biology.

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FIGURE 11: — dzy RNAi leads to defects in the ventral epidermis and persistent deep segmental grooves. (A, B) Stage 9. (A) WT. (B) dzy RNAi. Note remaining fold (arrow) and gaps at some AP borders and TCJs (arrowheads, inset). (C) dzy RNAi, stage 10. Gaps in the ventral epidermis are evident (arrows). (D–G) Stage 11. (D) WT. Segment grooves become apparent (arrows), and tracheal pits invaginate (arrowheads). (E–G) dzy RNAi. Note deep folds (arrows) and gaps in the ventral epidermis (arrowheads). (H–J) dzy RNAi, stages 12 and 14. Note abnormally deep segmental grooves (arrows) and holes in the ventral epidermis (arrowheads). (K) Epidermal defects are more frequent and widespread in cnoM/Z null mutants. (L) cno∆RA mutants share the deep segmental groove phenotype.