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. 2023 Jan 6;14:108. doi: 10.1038/s41467-022-35732-1

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Inflammatory profile in Mfn1KD myoblasts upon ODN2088 or Ru.521 treatment (1 µM, 24 h) (n = 4). b Inflammatory profile in Mfn2KD myoblasts upon ODN2088 or Ru.521 treatment (1 µM, 24 h) (n = 4). c Inflammatory profile in Fis1KD myoblasts upon ODN2088 or Ru.521 treatment (1 µM, 24 h) (n = 8). d Inflammatory profile in Drp1KD myoblasts upon ODN2088 or Ru.521 treatment (1 µM, 24 h) (n = 7). e Inflammatory profile upon Vdac1, Bax, Ppid, or DNaseIIa acute downregulation in Scr and Mfn1KD myoblasts (n = 4). a–e Two-way ANOVA test and post-hoc t tests. Data are expressed as mean of n independent experiments ± SEM.*p vs. Scr + vehicle <0.05 and ^#p vs. cognate KD + vehicle <0.05 in a–d; *p vs. Scr + siCtrl <0.05 and ^#p vs. cognate KD + siCtrl <0.05 in e. a–e Source data is provided in the Source Data File.