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. 2023 Jan 7;16:5. doi: 10.1186/s13048-022-01088-9

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Construction of a prognostic risk model on the basis of these differentially expressed GTs. A Univariate Cox regression analysis of GTs. Factors with P < 0.1 are shown. B-D The process of establishing a risk signature with six glycosyltransferase genes. Coefficients were calculated by multivariate Cox regression by LASSO. E The distribution of risk scores in the prognostic model. F The distribution of survival status in the prognostic model. Patients in the high-risk group had more deaths and a shorter survival time than those in the low-risk group (the right side of the dotted line). G The proportion of deaths in two groups. *P < 0.05