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. Author manuscript; available in PMC: 2023 Jan 8.
Published in final edited form as: Exp Eye Res. 2022 Sep 2;224:109213. doi: 10.1016/j.exer.2022.109213

Fig. 3: The absence of CCL2 delayed A-wave decline during B. cereus infection.

Fig. 3:

Eyes of C57BL/6J and CCL2−/− mice were infected with 100 CFU B. cereus. Retinal function was assessed by ERG at 8, 10, 12, 14 and 16 hours postinfection. In infected CCL2−/− eyes, retained A- wave responses were significantly greater than retained A- wave responses in infected C57BL/6J eyes at 12 (P=0.0348) and 14 (P=0.0308) hours postinfection (Fig 3A). B-wave responses in CCL2−/− eyes were significantly greater than the B-wave responses in the infected C57BL/6J eyes at 10 hours postinfection (P=0.0156, Fig 3B). Values represent means ± SEM of n ≥ 6 eyes at each time point with at least 3 independent experiments. *P ≤ 0.05 and nsP≥0.05.