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A
The list of ubiquitination (Ub)‐related proteins that interact with IRF7 according to the MS results.
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B
HEK293T cells were transfected with IRF7 and the indicated ubiquitin‐related expression plasmid. IB was performed to detect FLAG, followed by IP with an anti‐hyaluronic acid (HA) antibody.
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C, D
HEK293T cells were transfected with IFN‐β luciferase reporters and the indicated expression plasmids. Luciferase assays were performed to determine the fold changes with respect to the empty vector group.
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E
qRT–PCR analysis of IFN‐I in WT and KLHL22‐KO MEFs stimulated with polyI:C or SeV. The data are presented as the fold change relative to the Actb mRNA level.
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F
Immunoblot analysis of SeV‐induced IRF7 in whole‐cell lysates (WLs) of WT or KLHL22‐KO MEFs.
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G
After treatment with MG132, IRF7 was isolated by IP from WLs of KLHL22‐KO MEFs and subjected to IB using anti‐K48‐ubiquitin. Total cell lysates were also subjected to direct IB.
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H
HEK293T cells were transfected with IRF7 and FLAG‐K48R‐ubiquitin or FLAG‐ubiquitin in the presence (+) or absence (−) of KLHL22 expression plasmids. HA‐tagged IRF7 was isolated by IP, and the ubiquitination level was then measured by IB.
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I
HEK293T cells were transfected with IRF7 and distinct KLHL22 truncation mutants. IB of FLAG was performed, followed by IP with an anti‐HA antibody.
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J
HEK293T cells were transfected with KLHL22, FLAG‐K48‐ubiquitin, and various IRF7 point mutants. After treatment with MG132, IB of K48‐Ub was performed, followed by IP with an anti‐HA antibody.
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K
HEK293T cells were transfected with IFN‐α4 luciferase reporters and the indicated expression plasmids. Luciferase assays were performed to determine fold changes with respect to the empty vector group.