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High expression of MHC II, ILC2 may function as APC, promoting Th2/Th1 balance to Th2 shift in AECOPD patientsAECOPD patients with inflammation have rapidly deteriorated lung function, ILC2s which highly express MHC II are likely to perform APC-like functions, promoting the secretion of IL-4, IL-5 and IL-13 through the interaction of MHC II-like molecules with T-cell receptors, leading to elevated differentiation of Th2 cells and ultimately resulting in Th1/Th2 balance to Th2 shift.
