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. 2023 Jan 3;7(1):e0004. doi: 10.1097/HC9.0000000000000004

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Copyright © 2023 The Authors. Published by Wolters Kluwer Health, Inc. on behalf of the American Association for the Study of Liver Diseases.

This is an open access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0/

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THY1 may play profibrogenic roles in the liver fibrosis of biliary atresia. (A) Gene Ontology (GO) analysis of upregulated genes in patients with higher THY1 expressions. (GSE46960). (B) GO analysis of upregulated genes in THY1 positive versus negative cells in HSCs and myofibroblasts (GSE136103). (C) Immunofluorescence of fibrotic markers in the THY1⁺ cells isolated from biliary atresia (BA) livers using magnetic activated cell sorting. (D) The green fluorescent protein fluorescence and cell morphology of primary HSCs transfected with THY1 overexpression lentivirus.(E) Western blot of fibrotic markers in primary HSCs transfected with THY1 overexpression lentivirus. (F) Statistic analysis of western blot results in (E). *p < 0.05, ***p < 0.001.