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. 2023 Jan 10;15:2. doi: 10.1186/s13073-022-01145-4

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Mesangial cells exhibit dominant signaling networks in DN glomeruli. a Dot plot showing mesangial marker genes encoding transmembrane proteins responsible for the perception and transduction of mechanical signals. MSCs, mechanosensitive ion channels; GPCRs, G protein-coupled receptors. b Change of cell–cell interactions in all pairs of kidney cell types in DN vs. Ctrl mice. c Change of cell–cell interactions in all pairs of glomerular cell types in DN vs. Ctrl mice. d Increased cell–cell interactions for mesangial cell signaling to podocytes and glomerular endothelial cell in DN mice