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. 2022 Dec;10(24):1331. doi: 10.21037/atm-22-5148

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2022 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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MiR-21-3p destroyed the glycocalyx and induced inflammation in the THS rats treated with BRS. The levels of SDC-1, HPA1 (A), and IL-6, IL-1β, and TNF-α (B) in the serum of the BRS-treated THS rats transfected with lentivirus-mediated miR-21-3p were determined by their respective ELISA kits. *, P<0.05; **, P<0.01; ***, P<0.001, vs. sham. ^#, P<0.05; ^##, P<0.01; ^###, P<0.001, vs. model. ^&, P<0.05; ^&&, P<0.01; ^&&&, P<0.001, vs. BRS + miR-NC. THS, traumatic hemorrhagic shock; BRS, bicarbonate Ringer’s solution; LRS, lactate Ringer’s solution; NC, negative control; SDC-1, syndecan-1; HPA1, heparanase-1; IL, interleukin; TNF-α, tumor necrosis factor α.