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. 2023 Jan 16;23:7. doi: 10.1186/s12935-022-02836-z

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Identification of cuproptosis subtypes in PAAD. A Interactions among CRGs in PAAD. The size of the circle represents the impact of each CRG on the prognosis; the P value was evaluated by the log-rank test. Green dots represent protective factors, and violet dots represent risk factors. B Kaplan‒Meier curves of the three cuproptosis subtypes. C PCA identified three distinct subtypes based on the expression levels of CRGs. D Differences in the expression of CRGs among the three cuproptosis subtypes (***P < 0.001). E Survival analysis of the GSE21501 cohort with three cuproptosis subtypes