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. 2023 Jan 18;13:953. doi: 10.1038/s41598-023-28239-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Correlations between components of the classical and alternative RAS (PRA-S, Ang I, Ang II and Ang 1–7) and parameters of liver disease severity (MELD), severity of portal hypertension (HVPG), liver stiffness (LSM), endothelial dysfunction (vWF) and inflammation (IL-6). Correlations were assessed using Spearman’s Rho. ∗ denotes p values < 0.05, whereas ∗∗ indicates p values < 0.001. Ang angiotensin, HVPG hepatic venous pressure gradient, IL-6 interleukin-6, LSM liver stiffness measurement, MELD model for end-stage liver disease, RAS renin–angiotensin system, PRA-S plasma renin activity surrogate, vWF von Willebrand factor antigen.