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. 2023 Jan 21;8:42. doi: 10.1038/s41392-022-01265-8

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Immune escape by the BA.1 Omicron variant. a The ELISA assay shows the binding ability of 3A2A12, MM43, MM48 and mouse IgG to SARS-CoV-2 RBD (n = 4). b The neutralizing ability of 3A2A12 for SARS-CoV-2 and BA.1 Omicron pseudoviruses (n = 3), p < 0.001, determined by two-way ANOVA. c Inhibition of Delta and BA.1 Omicron pseudoviruses by convalescent patient sera (n = 10). d Inhibition of Delta (left) and BA.1 Omicron (right) pseudoviruses by COVID-19 convalescent patient sera in vaccinees (n = 6) and non-vaccinated group (n = 4). Data are represented as mean ± SEM, p-value was determined by the Wilcoxon rank sum test, (*p < 0.05, **p < 0.01, ***p < 0.001)