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. 2023 Jan 26;10:1. doi: 10.1186/s44156-022-00013-6

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — A–H Short axis basal, mid, apical and 4-chamber STIR images demonstrating subendocardial and with lesser degree midmyocardial and epicardial edema in different LV walls (mainly the apex) and also in RV walls (A–E), which has decreased compared with previous study (shown in Fig. 4A–C), indicative of partial improvement. Short axis post contrast views showing subendocardial, midmyocardial and epicardial LGE in anterior, septal and with lesser degree anterolateral LV walls, mainly consistent with residual inflammation and with lesser degree areas of midmyocardial scar formation in anterior and septal walls (F–H)