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. 2023 Jan 30;23:102. doi: 10.1186/s12885-023-10575-1

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 15 — CD276 expression is associated with the functions and treatment of GBM. A Single-cell analysis showed that CD276 is involved in regulating angiogenesis, proliferation, DNA damage and repair, EMT, inflammation, invasion and metastasis. B and C Data from the CancerSEA database showed that CD276 was correlated with various functions. D RT-qPCR was used to analyze CD276 and PCNA mRNA expression in patients with GBM compared with that in normal tissues. E and G The ROC plotter database was used to explore the relationship between CD276 expression and responses to chemotherapy in patients with GBM