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. 2023 Jan 4;27(2):44. doi: 10.3892/mmr.2023.12931

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Copyright: © Zhao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — OSR alleviates brain injury and neuronal apoptosis in I/R-induced rats. (A) Structure of OSR. (B) Pathological changes in rat brain tissue was assessed using hematoxylin and eosin staining. Scale bar, 25 µm. (C) Representative brain sections with the largest volume infarction were assessed using 2,3,5-triphenyltetrazolium chloride staining. (D) Infarct volume. (E) Apoptosis rate was assessed using TUNEL assay. Scale bar, 25 µm. (F) Protein expression levels of Bax and Bcl-2 were semi-quantified using western blotting. Data are presented as mean ± SD. Comparisons between multiple groups were performed using one-way ANOVA followed by Bonferroni's post hoc test for multiple comparisons. ***P<0.001. OSR, oxysophoridine; I/R, ischemia/reperfusion.