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. 2023 Jan 12;13:1096071. doi: 10.3389/fgene.2022.1096071

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Copyright © 2023 Lothion-Roy, Haigh, Harris, Metzler, Alsaleem, Toss, Kariri, Ntekim, Robinson, Khani, Gudas, Allegrucci, James, Madhusudan, Mather, Emes, Archer, Fray, Rakha, Jeyapalan, Rutland, Mongan and Woodcock.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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METTL3, METTL14, WTAP and CBLL1 expression in non-malignant and PCa tissue and genetic alteration analysis. Data from the GDC TCGA Prostate Cancer (PRAD) dataset were downloaded from USCS Xena [(A); n = 623]. METTL3, METTL14, WTAP and CBLL1 expression in non-malignant prostate tissue was compared with primary prostate adenocarcinoma specimens. The cBioPortal for Cancer Genomics was utilised to assess the incidence of amplification, deep deletion and mutation of METTL3, METTL14, WTAP and CBLL1 in patients with adenocarcinoma, metastatic adenocarcinoma and neuroendocrine PCa [(B); n = 1,059]. ns, not significant; **p ≤ .005, ****p ≤ .0001 by unpaired t-test.