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. 2023 Jan 20;33(2):147–164. doi: 10.1038/s41422-022-00760-5

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Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — hEnSCs attract MoMFs/Kupffer cells in ALF livers and secrete CST1 in a paracrine fashion to suppress the IFN-induced pro-inflammatory macrophage polarization, and to promote the reprogramming of MoMFs/Kupffer cells into anti-inflammatory phenotypes by IL10, which finely tunes the immune microenvironments to accelerate the resolution of inflammation.