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. 2022 Sep 24;29(1):52–62. doi: 10.1177/13524585221125369

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© The Author(s), 2022

This article is distributed under the terms of the Creative Commons Attribution 4.0 License (https://creativecommons.org/licenses/by/4.0/) which permits any use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — PRM-MS ratios of selected proteins for validation with PRM-MS measurements separated for clinical phenotype within mADS group in ADEM and non-ADEM patients.

CSF samples of adult controls (n = 16) were pooled and included for technical QC.

Using Wilcoxon rank sum test: *p < 0.05, **p < 0.01, and ***p < 0.001.

ADEM: acute disseminated encephalomyelitis; CPE: Carboxypeptidase E; mADS: monophasic acquired demyelinating syndrome; MEGF8: Multiple epidermal growth factor-like domains protein 8; NEGR1: Neuronal growth regulator 1; NUCB1: Nucleobindin-1; POMS: pediatric-onset multiple sclerosis; SEMA7A: Semaphorin-7A; QC: quality control.