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. 2023 Feb 2;21:29. doi: 10.1186/s12964-022-01022-y

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Impaired inflammatory response with SF3A2 overexpression after I/R. A Heat mapping of the top 20 ranked (by p value and fold-change) genes that were differentially expressed in H9c2 cells transfected with SF3A2 overexpression plasmid, subjected to I/R. B Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analysis of differentially expressed genes in H9c2 cells transfected with SF3A2 overexpression plasmid subjected to I/R. C–F mRNA gene expression analysis of inflammatory gene was verified in cell lysates and supernatants from H9c2 cells subjected to I/R, transfected with vector or SF3A2 overexpression plasmid. Data are expressed as mean ± SD (n = 3). *P < 0.05 vs. control+ I/R group