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. 2023 Feb 1;15(1):2168992. doi: 10.1080/19490976.2023.2168992

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© 2023 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 1. — Possible mycobiome-related mechanisms of action in Irritable Bowel Syndrome. (a) Cellular and molecular mechanisms through which host and mycobiome may interact. Part of these interactions were described in IBS and IBS-like rodent models. Others were addressed in the somatic pain field and still need confirmation in IBS (see text). BLP, balloon-like protrusion; CGRP, calcitonin-related gene peptide; CRF, corticotropin-releasing factor; H1R, histamine-1-receptor; PAR2, protease activated receptor 2; PKC, protein kinase C; PLC, phospholipase C; TRPV1, transient receptor potential channel vanilloid 1. (b) Recent observations and considerations regarding the gut mycobiome in IBS. (c) Possible strategies for modulation of the fungal community in patients with IBS. Illustration created with BioRender.com.