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A] THC modulates CB1R and CB2R that are endogenously activated by 2-AG and AEA, and GLRA3 receptors that are endogenously activated by glycine, as demonstrated by in vivo genetic and antagonist experiments. B] THC modulates GPR55 that is endogenously activated by lysophosphatidyl inositol (LPI) and 5-HT3A receptors that are endogenously activated by serotonin, as suggested by cell culture experiments.
