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. 2023 Feb 3;13:1983. doi: 10.1038/s41598-023-27981-x

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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IL-10 promoted repair site arteriole formation. TA muscle cross-sections prepared from 56 DPI were individually stained for (a) CD31 (green) or (b) α-SMA (red). Scale bar = 100 µm. Immuno-stained tissue cross sections prepared from 56 DPI tissue samples were quantified to determine (c) capillary density (capillaries/mm²), and (d) arteriole density (arterioles/mm²). (e) VEGF and (f) IL-6 concentrations (pg/mg) were measured from 14 DPI tissue samples using multiplex ELISA. Group means + SD are presented; n = 4–5/group. The *, **, and *** indicate statistically significant difference of p < 0.05, p < 0.01, and p < 0.001 between groups.