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. 2023 Jan 31;67:10.29219/fnr.v67.9024. doi: 10.29219/fnr.v67.9024

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© 2023 Xiaoping Yu et al.

This is an Open Access article distributed under the terms of the Creative Commons Attribution 4.0 International License, allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material for any purpose, even commercially, provided the original work is properly cited and states its license.

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Fig. 3 — Genistein inhibited the AKR1C3 expression in mouse xenograft tumors and CRPC cell line. (a) Expression of AKR1C3 in tumor tissues of mice after genistein treatment. (b) Quantitative polymerase chain reaction analyses of mRNA expression in control and genistein-treated. Data are presented as the means ± SD. * P < 0.05 vs. control. (c) Protein expression of AKR1C3 in VCaP and 22RV1 cells after genistein treatment. Data represent means ± SD of three independent experiments. *P < 0.05 vs. control.