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. 2023 Feb 6;14(2):86. doi: 10.1038/s41419-023-05631-4

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A, B FcγRIII and FcγRII on peripheral MDSCs (HC n = 13, SS n = 13) were detected by flow cytometry. Representative flow cytometric analysis and percentages of FcγRIII⁺ MDSCs and FcγRII⁺ MDSCs and the MFI of FcγRIII and FcγRII on MDSCs from HC and SS patients were shown. C–H MDSCs or IgG-preprocessed MDSCs were stimulated with or without IgG or HAIG for 24 h and then co-cultured with CFSE-labeled CD4⁺ T cells for 72 h. Representative flow cytometric analysis and percentages of CD4⁺ T cell subsets were shown. C CD4⁺IFN-γ⁺ Th1 cells, D CD4⁺IL-4⁺ Th2 cells, E the ratio of Th1/Th2, F CD4⁺IL-17A⁺ Th17 cells, G CD4⁺Foxp3⁺ Treg cells, H the ratio of Th17/Treg. All the data are representative of two independent experiments. n = 4, *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001.