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. 2022 Sep 10;26(1):193–196. doi: 10.1007/s10456-022-09855-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Attenuation of liver cirrhosis and portal hypertension by the combination treatment. Typical cirrhotic appearance with extensive nodular formation (a) and fibrotic septa (b) was presented in livers of the TAA group. These hepatic nodules and fibrotic septa (Masson’s trichrome staining, × 100 magnifications) were almost not observed in the TAA + combination group. The fibrotic areas (c) and hepatic α-SMA (d) and collagen III (e) mRNA quantified by quantitative real-time PCR (qRT-PCR) also decreased in the TAA + combination group. Mean arterial pressure (f) and heart rate (g) were comparable in three groups. Portal pressure in the TAA group was the highest among three groups (h). *p < 0.05 versus control group; ^#p < 0.05 versus TAA group