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. 2023 Feb 8;15:44. doi: 10.1007/s40820-023-01018-4

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 16 — Schematic illustration of a fabrication of the nanosonosensitizer P/M@CasMTH1 and b US-triggered genome-editing-augmented SDT for tumor treatment. c Confocal laser scanning microscopy (CLSM) images of A549-EGFP cells after different treatments. d Western blot analysis of the MTH1 expression in A549 cells after different treatments. e NGS analysis of indel percentage in P/M@CasMTH1 or P/M@CasMTH1 + US treatment groups. f Viability of A549 cells after SDT or combined therapy treatment. g Digital photographs of dissected tumors in different treatment groups [181].

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