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. 2023 Feb 15;13:32. doi: 10.1186/s13578-023-00979-8

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© This is a U.S. Government work and not under copyright protection in the US; foreign copyright protection may apply 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Global analysis of GLIS3, NKX2.1, and PAX8 genomic binding in mouse thyroid glands. A–C HOMER analysis, heatmap, and ChIP-Seq read density of GLIS3 (A), NKX2.1 (B), and PAX8 (C) binding data. Heatmap of the 2 kb region is centered on each of the binding peaks identified. D Genomic context of the GLIS3, NKX2.1, and PAX8 peaks within the whole mouse genome (mm10)