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. 2023 Feb 16;23:160. doi: 10.1186/s12885-023-10639-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — LINC02154 immune microenvironment associated analysis and drug sensitivity analysis. A Relationship between LINC02154 expression type and immune molecular typing. B Analysis of immune function in high LINC02154 and low LINC02154 expression groups. C Correlation analysis between LINC02154 and positive markers related to immunotherapy. D Analysis of the correlation between LINC02154 and critical steps of the cancer immunity cycle. E Immune checkpoint analysis of high LINC02154 and low LINC02154 expression groups. F-L Susceptibility analysis was performed separately for axitinib, doxorubicin, gemcitabine, pazopanib, sorafenib, sunitinib, and temsirolimus in high LINC02154 expression group and low LINC02154 expression group