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. 2023 Feb 1;12:e82283. doi: 10.7554/eLife.82283

Table 2. Comparison of human MFN2R707W-associated lipodystrophy with phenotype of Mfn2R707W/R7007W mice.

BAT, brown adipose tissue; GTT, glucose tolerance test; HFD, high-fat diet; ISR, integrated stress response; MFN2, mitofusin 2; mRNA, messenger ribose nucleic acid; mtDNA, mitochondrial DNA; PLIN1, perilipin 1; TEM, transmission electron microscope; WAT, white adipose tissue.

Human Phenocopy? Mouse
Upper body adipose overgrowth No No difference in weight of any adipose depots
Lower limb lipoatrophy No
Insulin resistance No No difference on GTT or fasting insulin
Lower serum leptin concentration Yes Seen on chow or HFD; lower secretion seen from adipose explants
Severely reduced adipose leptin mRNA expression Partial Modest decrease only; only significant in some analyses
Lower serum adiponectin concentration Yes Seen on chow or HFD; lower secretion seen from adipose explants
Severely reduced adipose adiponectin mRNA expression Yes Seen on chow or HFD and in adipose explants
No change in WAT MFN2 protein expression (only over-grown WAT studied) Yes No difference in any tissue studied, though variable in WAT and BAT.
No difference in adipocyte size Yes True on both chow and HFD
Disorganised, fragmented WAT mitochondria on TEM Partial More circular mitochondria with trend towards reduced cristae
Upregulation of nuclear and down regulation of mitochondrial Oxphos transcriptional pathway Yes True in inguinal and epididymal WAT, and BAT
Lower Oxphos complex II and III but preserved complex I and IV protein Partial Lower complex I and IV protein in WAT
Lower WAT mtDNA Partial Lower mtDNA in BAT but not WAT
Transcriptional activation of ISR Yes Seen in inguinal and epididymal WAT, and BAT