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. 2023 Feb 18;11:20. doi: 10.1186/s40364-023-00458-9

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Common changes of hepatic transcripts due to differential diet intake, ages, and FXR KO in association with the expression of genes implicated in human HCC patient survival. A Venn diagram shows the numbers of common genes (transcripts) (fold change ≥2 and adjusted p value < 0.05). B A heatmap reveals transcript levels of mouse genes implicated in hepatic metabolic disease (n = 76) and human genes differentially expressed in human HCC and healthy livers. C Pathway enrichment analyses of 76 mouse hepatic genes that are implicated in human HCC survival using Metascape (adjusted p value < 0.05). The numbers of differentially expressed genes in each pathway are shown in the bar graph. LIHC, liver hepatocellular carcinoma