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. 2023 Feb 21;14:10. doi: 10.1186/s13293-023-00493-z

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — HFD regulates cochlear AdipoR1, COX IV, HO-1 and G3bp1; and liver AdipoR1. Cochlear AdipoR1, cytochrome c oxidase subunit IV (COX IV) heme oxygenase-1 (HO-1; also known as heat shock protein 32, HSP 32), and a marker of stress granule (G3bp1) levels were measured in the whole cochlear lysates by Western blot analysis (a) and quantified (b–e). Cochlear AdipoR1 and HO-1 presented main effects of sex and diet while COX IV and G3bp1 displayed a main effect of diet. f Liver AdipoR1 protein levels were measured by Western blotting (a) and quantified (g). Female AdipoR1 levels were regulated by diet, but no changes were seen in male AdipoR1 in the liver. Different letters indicate significant differences between treatments. Two-way ANOVA with Tukey’s multiple comparisons test. p < 0.05. ^amain effect of sex; ^bmain effect of diet; ^cinteraction effect