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. 2023 Feb 22;13:38. doi: 10.1186/s13578-023-00985-w

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Glutamine metabolism promotes migration, invasion, and cytoskeleton morphology by up-regulating ARPC1A expression in PCa cells. A GSEA analysis revealed a significant positive association between high ARPC1A expression and glutamine metabolism. B Proposed model describing the roles of glutamine metabolism and ARPC1A in PCa. C–E ARPC1A expression, cytoskeletal morphology, and the migration and invasion potential of PC-3 cells cultured in different types of media. *: P < 0.05; ns no statistical significance, Gln glutamine, Glc Glucose, DKG dimethyl α-ketoglutarate. F–H ARPC1A expression, cytoskeletal morphology, and the migration and invasion potential of PC-3 cells under the indicated treatment conditions. *: P < 0.05; ns no statistical significance