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. 2023 Feb 22;14:1005. doi: 10.1038/s41467-023-36694-8

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a, b Transmission electron microscopy of phage FD1 and FD1 bound to its bacterial target (b). In addition, images of all phages are presented in Supplemental Fig. S2a. This experiment was done twice (c) FD1 plaques on Cutibacterium acnes strain 27. Note the diverse sizes of plaques, although they originate from a single plaque. d The CM-resistant strain 28, growth with CM, phage FD1, and combination. The results are the average of triplicates, presented as mean ± standard deviation (SD). e FD1-resistant strain 21 growth with CM, FD1 phage, and their combination. The two lower curves, CM and CM + FD1, overlap. The results are the average of triplicates, presented as mean ± SD. CM, clindamycin. Source data are provided as a Source Data file.