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. 2023 Feb 23;24:14. doi: 10.1186/s12868-023-00782-8

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© The Author(s) 2023, corrected publication 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — IL-17 induces neuronal toxicity in primary hippocampal neurons. Mouse primary hippocampal neurons were treated with DMSO for the control group and IL-17 for the IL-17 group for 9 d. Changes in neuronal morphology were measured by immunofluorescence staining with anti-PSD95 (red), anti-MAP2 (green) antibodies, and co-labelled with DAPI (blue). Representative images (20×magnification) after treatment (A), Sholl analysis (B), quantitative analyses of dendritic length (C), N = 20 hippocampal neurons. Data are presented as the mean ± SD. p value significance is calculated from a T-test. ***p < 0.001 vs the control group. For western blotting original images, please see Additional file 1, and Adobe Photoshop software (2021) was used to cut the parts of interest from the original images