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. 2023 Feb 19;15(4):1319. doi: 10.3390/cancers15041319

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© 2023 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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AhR is involved in the function of SRF in carcinogenesis. (A) Migration and invasion assays on SAS and HSC-3 cells after adding the AhR inhibitor PDM2. Immunofluorescence staining of AhR (B), E-cadherin (C), and N-cadherin (D) in SAS and HSC-3 cells after adding PDM2. The scale bar is shown in lower-right corner. (E) Western blotting results of AhR, E-cadherin, and N-cadherin in SAS and HSC-3 cells after adding PDM2. Original western blots are shown in Figure S9. AhR, aryl hydrocarbon receptor; E-cad, E-cadherin; N-cad, N-cadherin; OSCC, oral squamous cell carcinoma; PDM2, AhR inhibitor; SRF, serum response factor.