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. 2023 Feb 10;9(2):e13617. doi: 10.1016/j.heliyon.2023.e13617

Fig. 8.

Fig. 8

Correlationship between GPR65 expression and tumor immune microenvironment and infiltration. (A) Connection between the expression of GPR65 and immune stimulatory and inhibitory. (B) Correlation between the expression of GPR65 and chemokine, chemokine receptor, MHC, immunostimulator, and immunoinhibitor. (C) Relationship between GPR65 expression and stromal score. (D) Correlation of GPR65 with B cell, CD4 T cells, CD8 T cells, neutrophil, macrophage, and DC infiltration levels using the TIMER database. (E) Correlation of GPR65 with infiltration levels of B cells, CAFs, CD4 T cells, CD8 T cells, endothelial cells, macrophages, and NK cells using the EPIC database. (F) Correlation between GPR65 and infiltration levels of B cells, M1 macrophage, M2 macrophage, Monocytes, neutrophils, NK cells, CD4 T cells, CD8 T cells, Tregs, and dendritic cells using the quanTIseq database.