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. 2023 Feb 4;24(4):3116. doi: 10.3390/ijms24043116

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© 2023 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Reduced in vivo occlusion time in Mgl^−/− mice. Plasma TXB2 and vWF concentrations were measured by ELISA (mean ± SEM) in plasma from (A,C) Mgl^−/− and (B,D) platMgl^−/− mice (n = 6). Thrombus formation was induced by (E) applying a drop of 10% FeCl₃ in Mgl^−/− mice and (F) after mechanical injury by a single firm compression with a forceps for 10 s in platMgl^−/− mice. Occlusion time was recorded using an ultrasonic flowprobe. Data are expressed as mean ± SEM (n = 5–7). *, p < 0.05.

Reduced in vivo occlusion time in Mgl^−/− mice. Plasma TXB2 and vWF concentrations were measured by ELISA (mean ± SEM) in plasma from (A,C) Mgl^−/− and (B,D) platMgl^−/− mice (n = 6). Thrombus formation was induced by (E) applying a drop of 10% FeCl₃ in Mgl^−/− mice and (F) after mechanical injury by a single firm compression with a forceps for 10 s in platMgl^−/− mice. Occlusion time was recorded using an ultrasonic flowprobe. Data are expressed as mean ± SEM (n = 5–7). *, p < 0.05.