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ILC plasticity in response to gut and genital tract infections. Salmonella and Campylobacter species induce IL-12 production by accessory cells to drive conversion of ILC3s toward IFNγ-producing ILC1s in the intestine by downregulating RORγt and upregulating T-bet expression. Similarly, Chlamydia drives ILC3→ILC1 conversion to initiate IFNγ-mediated protective responses.
