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. Author manuscript; available in PMC: 2023 May 12.
Published in final edited form as: Pediatr Res. 2022 Aug 30;93(6):1539–1545. doi: 10.1038/s41390-022-02278-3

Figure 5: Hemodynamic changes with respiratory arrest.

Figure 5:

(A) PIVA f1 amplitude from a saphenous venous waveform increased from 0.07 mmHg (IQR=0.06 to 0.23) to 0.38 mmHg (IQR=0.21 to 0.49), p=0.01, during respiratory arrest. After recovery from respiratory arrest, the f1 amplitude returned to near baseline levels 0.08 mmHg (IQR=0.04 to 0.23), p= 1.0 (vs baseline). * indicates p<0.05, ** indicates p<0.01. (B) CVP did not change significantly from baseline 9.3 mmHg (SD=6.0) during arrest 10.5 mmHg (SD=4.8) nor after recovery from arrest 8.6 mmHg (SD=3.6), p=0.91 and 0.81, respectively. (C) MAP significantly decreased during respiratory arrest from 119 mmHg (SD=23) to 67 mmHg (SD=22), p=0.004, then recovered to near baseline values at 136 mmHg (SD=33), p=0.5 (vs baseline). ** indicates p<0.01. (D) HR decreased during respiratory arrest from 341 bpm (IQR=297 to 379) at baseline to 129 bpm (IQR=114 to 220), p=0.0004. Recovery HR at 285 bpm (IQR=275 to 318) was statistically indistinguishable from baseline, p=0.18. ** indicates p<0.01.

Abbreviations: PIVA, peripheral intravenous waveform analysis; CVP, central venous pressure; MAP, mean arterial pressure; HR, heart rate; bpm, beats per minute.