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. 2023 Feb 27;18:6. doi: 10.1186/s13062-023-00361-1

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — miRNA-205 was involved in the ADSC-Exos-mediated promotion of the proliferation of microvascular endothelial cells. A. The level of miR-205 in ADSCs-exosomes was evaluated by metabolomics analysis; B. The level of miR-205 in ADSCs-exosomes was determined by RT-qPCR; C. ADSC cells treated with cy3 labeled miR-205 were co-cultured with microvascular endothelial cells. D. Cy3 labeled miR-205 (red) were present in microvascular endothelial cells. E. Relative level of miR-205 in ADSCs-exosomes was determined by RT-qPCR. Data were presented as Mean ± SEM, n = 6 independent experiment. ^**P < 0.05