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. 2022 Dec 19;8(1):205–210. doi: 10.1002/epi4.12678

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© 2022 The Authors. Epilepsia Open published by Wiley Periodicals LLC on behalf of International League Against Epilepsy.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Serial T2‐weighted axial MRI and FDG‐PET images (radiological view) demonstrating the left basal ganglia abnormalities. MRI at age 1.6 (A), 2.1 (B), 2.4 (C), 2.6 (D), 3.9 (F), and 10 (H) years demonstrate initial progression then stability of the left basal ganglia abnormality, maximal in the caudate head. Tracts from the first (short arrow) and second (long arrow) biopsies are evident in (D, F, and H). FDG‐PET at age 3.1 years (E) shows normal metabolism in both basal ganglia and thalami, and relative hypometabolism in the left hemisphere. FDG‐PET at age 6.3 years (G) shows marked hypometabolism in the left basal ganglia and thalamus.