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. 2022 Dec 20;1(1):e000277. doi: 10.1136/bmjmed-2022-000277

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© Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution 4.0 Unported (CC BY 4.0) license, which permits others to copy, redistribute, remix, transform and build upon this work for any purpose, provided the original work is properly cited, a link to the licence is given, and indication of whether changes were made. See: https://creativecommons.org/licenses/by/4.0/.

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Associations of genetically proxied lipid lowering and antihypertensive therapies with hepatic fat. The associations for genetically proxied lipid lowering target was scaled to a decrease of 10 mg/dL low density lipoprotein cholesterol concentrations. The associations for genetically proxied antihypertensive target was scaled to a decrease of 10 mm Hg systolic blood pressure. The x axis unit is standard deviation (SD) change in liver fat percentage. One SD increase is approximately a 4.25 unit increase of absolute liver fat percentage points. CI=confidence interval; HMGCR=3-hydroxy-3-methyl-glutaryl-coenzyme A reductase; LDLR=low density lipoprotein receptor; NPC1L1=Niemann-Pick C1-like 1; PCSK9=proprotein convertase subtilisin/kexin type 9