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. 2023 Mar 3;21:48. doi: 10.1186/s12964-023-01065-9

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — ARID1A serves as a protective factor in lung adenocarcinoma with or without EGFR mutations. A Lung adenocarcinoma patients harboring ARID1A mutations had poorer overall survival than the wild-type group. B–D Online database analysis revealed that low ARID1A expression was associated with poor overall survival in LUAD patients. E Low ARID1A expression was associated with a poor prognosis in EGFR-mutant lung adenocarcinoma patients in the TCGA database. F Verification of the relative expression of target proteins after the construction of ARID1A knockdown EGFR-mutant lung adenocarcinoma cell lines