Table 6:
Author | Sample Model | Metabolites/Metabolic Pathways of Importance |
---|---|---|
Thompson et al., 2011 14 | Human | This study did not report metabolites, and instead was included as a reference as it reported daily variations of serum amino acid and acylcarnitine levels. |
Maher et al., 2012 19 | Ovine | BCAAs(↓), 3-Methylhistidine(↑), Glycine(↑), Creatinine(↑), Creatine(↑) at 4 weeks and 12 weeks in post-ACL Transection OA model versus HC. |
Senol et al., 2019 23 | Human | Phospholipid species(↑), Indoleacetic acid(↑), Urea(↑), Valine(↑), Alanine(↑), FA metabolites(↑) increased in OA versus Control. |
Tootsi et al., 2018 26 | Human | Total Acylcarnitine species to Carnitine species ratio was decreased in OA versus HC. Carnitine Palmitoyltransferase 1 activity was decreased in OA versus HC. |
Maerz et al., 2018 34 | Rat | Acylcarnitine species increased at 72 hours, 4 weeks, and 10 weeks post-ACL rupture OA model versus HC. Most significantly affected pathways in OA versus HC identified as cyanoamino acid metabolism, methane metabolism, sphingolipid metabolism, histidine metabolism, glutathione metabolism, tryptophan metabolism, beta-alanine metabolism, fructose and mannose metabolism, and amino and nucleotide sugar metabolism. |
Chen et al., 2018 36 | Human | BCAAs(↑), Tryptophan(↑), Alanine(↑), 4-Hydroxy-L-Proline(↑), Creatine(↑), Arginine(↑), Lysine(↑), Tyrosine(↑), Glutamine (↓), Phenylalanine(↓), Serine(↓), Proline(↓), Gamma-Aminobutyric Acid(↓), Creatinine(↓), Taurine(↓), Asparagine(↓), Acetyl-Carnitine(↓), Citrulline(↓) in OA versus HC. |
Zhai et al., 2010 38 | Human | Increased BCAA to Histidine ratio in OA versus HC, with ratio of Valine to Histidine and Isoleucine to Histidine having strongest statistical significance. |
Zhang et al., 2015 39 | Human | CRP(↑), Homocysteine(↑), Tryptophan(↑), Glycine(↓), Histidine(↓) in OA versus HC. |
Zhao et al., 2021 44 | Rat | L-Tryptophan(↑), Gamma-Aminobutyric Acid(↑), Carbamic Acid(↑), L-Carnitine(↑), Stearic Acid(↑), L-Arginine(↓) in OA versus HC. |
Wallace et al., 2022 45 | Mouse | Day 1 Following Injury-Induced OA: Arginine Synthesis(↑), Proline Degradation(↑), Nicotine Degradation(↑), Phospholipase Pathway(↓) in OA versus HC. Day 8 Following Injury-Induced OA: Tyrosine Degradation(↑), |
Phospholipase Pathway(↑), Nicotine Degradation(↓) in OA versus HC. | ||
Xiao et al., 2022 48 | Rabbit | ɛ, ɛ, ɛ Trimethyllysine(↓), Ascorbic acid(↑), Otonecine(↑), and Tranexamic Acid(↓) in OA versus HC. BCAA Biosynthesis and Degradation (Leucine-Valine, L-(+)-Valine)(↑), HIF-1 Pathway(↑), Antioxidant Metabolism (Glutathione and Ascorbic Acid)(↑), Phenylalanine Metabolism(↑), Pantothenate and CoA Biosynthesis(↑), Tyrosine Metabolism(↓) in OA versus HC. |
Pertusa et al., 2022 49 | Human | Amino Acid Metabolism (Alanine, Glycine, Phenylalanine, and Tyrosine), Lactate, Acetate, Phosphocholine, Bacterial Co-Metabolism (2-Aminobutyrate, 4-Aminobutyrate, N(CH3)3, Dimethylamine) significantly altered in OA versus HC following adjustment for age, BMI, and bone mineral density. |
Key: (↑) indicates an increase in concentration in OA compared to controls, (↓) indicates a decrease in concentration in OA compared to controls.