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. 2023 Feb 22;11:1104126. doi: 10.3389/fbioe.2023.1104126

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Copyright © 2023 Salehi, Naghib, Garshasbi, Ghorbanzadeh and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Synthesis of UCCy@Gel for acute MI prevention (dark phase) and therapy (light phase): the UCCy@Gel consumes oxygen via respiration to generate a hypoxic microenvironment, resulting in the upregulation of HSP70, which enhanced the tolerance of CMs for MI (left); Under 980 nm NIR irradiation, the UCCy@Gel releases photosynthetic oxygen through UCL to inhibit macrophage M1 polarization and downregulated proinflammatory cytokines IL-6 and TNF-α to repair myocardial injury (right) (Liu Y. et al., 2022).