Nutrients Suppress Phosphatidylinositol 3-Kinase/Akt Signaling via Raptor-Dependent mTOR-Mediated Insulin Receptor Substrate 1 Phosphorylation -- Tzatsos and Kandror 26 (1): 63 Data Supplement - Supplemental material -- Molecular and Cellular Biology

Nutrients Suppress Phosphatidylinositol 3-Kinase/Akt Signaling via Raptor-Dependent mTOR-Mediated Insulin Receptor Substrate 1 Phosphorylation
Mol. Cell. Biol. Tzatsos and Kandror 26: 63

Supplemental material

Files in this Data Supplement:

Supplemental file 1 - Fig. S1 (Glucose stimulates IRS-1 phosphorylation on Ser636/639 in an insulin-independent manner), S2 (Metformin suppresses mTOR signaling and enhances Akt phosphorylation in L6 myotubes), S3 (Metformin enhances Akt signaling), S4 (Overexpression of wild-type S6K1 does not interfere with IRS-1 phosphorylation on Ser636/639), and S5 (S6K1 [uppercase delta]2-46/[uppercase delta]CT104 mutant overexpression does not interfere with Rheb ability to stimulate IRS-1 phosphorylation on Ser636/639)
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